Fe_xN produced in pharmaceutical sludge biochar by endogenous Fe and exogenous N doping to enhance peroxymonosulfate activation for levofloxacin degradation

For preparing high performance biochar to be applicated in persulfate-based oxidation treatment of wastewater, the feasibility of deriving Fe-N biochar from pharmaceutical sludge by endogenous Fe and exogenous N doping was investigated. With exogenous urea doping, Fe_xN contained biochar (PZBC800U) was successfully derived from endogenous Fe(OH)₃ contained pharmaceutical sludge. PZBC800U effectively activated peroxymonosulfate (PMS) to remove 80 mg·L⁻¹ levofloxacin (LEV) within 90 min. The main mechanism of PMS activation by PZBC800U for LEV degradation was revealed as non-radical pathways dominated by ¹O₂ generation and direct electron transfer. The formation of Fe_xN combined with the increase of pyridinic-N in the biochar changed the electronic structure, improved the electron transfer ability, and thus achieved the excellent PMS activation capacity of the biochar. The vital function of endogenous Fe(OH)₃ was verified by comparing PZBC800U to Fe leached and extra Fe added controls. A total of 18 intermediates in the degradation of LEV were identified, and degradation pathways were proposed. Combined with the average local ionization energy calculation, the priority of piperazine breakage during LEV degradation was experimentally proved and mechanistically elucidated. This study provides a new insight into Fe_xN biochar preparation from pharmaceutical sludge and the mechanisms of its excellent PMS activation performance for LEV degradation.