Early visual processing deficits in dysbindin-associated schizophrenia.

GARY DONOHOE

doi:10.1016/j.biopsych.2007.07.022

Early visual processing deficits in dysbindin-associated schizophrenia.

GARY DONOHOE

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60 Citations (Scopus)

Abstract

Variation at the dysbindin gene (DTNBP1) has been associated with increased risk for schizophrenia in numerous independent samples and recently with deficits in general and domain-specific cognitive processing. The relationship between dysbindin risk variants and sensory-level deficits in schizophrenia remains to be explored. We investigated P1 performance, a component of early visual processing on which both patients and their relatives show deficits, in carriers and noncarriers of a known dysbindin risk haplotype.

Original language	English (Ireland)
Pages (from-to)	484-489
Number of pages	6
Journal	Biological Psychiatry
Volume	63
Issue number	5
DOIs	https://doi.org/10.1016/j.biopsych.2007.07.022
Publication status	Published - 1 Mar 2008

Keywords

Dysbindin
EEG
ERP
P1
schizophrenia
sensory processing

Authors (Note for portal: view the doc link for the full list of authors)

Authors
Donohoe G, Morris DW, De Sanctis P, Magno E, Montesi JL, Garavan HP, Robertson IH, Javitt DC, Gill M, Corvin AP, Foxe JJ

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10.1016/j.biopsych.2007.07.022

Cite this

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abstract = " Variation at the dysbindin gene (DTNBP1) has been associated with increased risk for schizophrenia in numerous independent samples and recently with deficits in general and domain-specific cognitive processing. The relationship between dysbindin risk variants and sensory-level deficits in schizophrenia remains to be explored. We investigated P1 performance, a component of early visual processing on which both patients and their relatives show deficits, in carriers and noncarriers of a known dysbindin risk haplotype. ",

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AB - Variation at the dysbindin gene (DTNBP1) has been associated with increased risk for schizophrenia in numerous independent samples and recently with deficits in general and domain-specific cognitive processing. The relationship between dysbindin risk variants and sensory-level deficits in schizophrenia remains to be explored. We investigated P1 performance, a component of early visual processing on which both patients and their relatives show deficits, in carriers and noncarriers of a known dysbindin risk haplotype.

KW - Dysbindin

KW - EEG

KW - ERP

KW - P1

KW - schizophrenia

KW - sensory processing

UR - https://www.scopus.com/pages/publications/38949126972

U2 - 10.1016/j.biopsych.2007.07.022

DO - 10.1016/j.biopsych.2007.07.022

M3 - Article

VL - 63

SP - 484

EP - 489

JO - Biological Psychiatry

JF - Biological Psychiatry

IS - 5

ER -

Early visual processing deficits in dysbindin-associated schizophrenia.

Abstract

Keywords

Authors (Note for portal: view the doc link for the full list of authors)

Access to Document

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